Тип публикации: статья из журнала

Год издания: 2005

Идентификатор DOI: 10.1007/s10517-006-0004-8

Ключевые слова: Apoptosis, Lipid peroxidation, Lymphocytes, Monocytes, Tuberculosis, reactive oxygen metabolite, tuberculostatic agent, adult, antibiotic resistance, antibiotic sensitivity, article, controlled study, female, human, human cell, intensive care, lung tuberculosis, lymphocyte, maintenance therapy, major clinical study, male, monocyte, peripheral blood mononuclear cell, respiratory burst, Antitubercular Agents, Humans, Leukocytes, Mononuclear, Middle Aged, Tuberculosis, Pulmonary

Аннотация: LPO and apoptosis in blood mononuclear cells were studied in patients with pulmonary tuberculosis before and during treatment with standard chemotherapeutics. Pulmonary tuberculosis was accompanied by LPO activation and intensification of apoptosis in lymphocytes and monocytes. These changes were observed before and after the coursПоказать полностьюe of intensive care. The intensity of lipid peroxidation retuned to normal, while activity of apoptosis remained high after therapy. © 2005 Springer Science+Business Media, Inc.

Журнал: Bulletin of Experimental Biology and Medicine

Выпуск журнала: Vol. 140, Is. 5

Номера страниц: 489-491

• Novitskii V.V. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Fed)
• Strelis A.K. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Feder)
• Tkachenko S.B. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Fed)
• Esimova I.E. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Feder)
• Shil'ko T.A. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Feder)
• Urazova O.I. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Feder)
• Voronkova O.V. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Fed)
• Sinitsina V.A. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Fed)
• Filinyuk O.V. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Fede)
• Zemlyanaya N.A. (Department of Pathophysiology, Central Research Laboratory, Siberian State Medical University, Tomsk, Russian Federation, Department of General Pathology and Pathophysiology, Russian Medical Academy for Postgraduate Education, Russian Fe)

• Scopus